CD 11 c+ Dendritic Cells Maintain Antigen Processing, Presentation Capabilities, and CD 4+ T-Cell Priming Efficacy Under Hypercholesterolemic Conditions Associated With Atherosclerosis by René R. S. Packard, Elena Maganto-García, Israel Gotsman, Ira Tabas, Peter Libby, and Andrew H. Lichtman Circulation Research Volume 103(9): 965 -973 October 24, 2008 Copyright © American Heart Association, Inc. All rights reserved.
Figure 1. CD 11 c+ cells accumulate lipids within atherosclerotic plaques and when exposed to cholesterol in vitro. René R. S. Packard et al. Circ Res. 2008; 103: 965 -973 Copyright © American Heart Association, Inc. All rights reserved.
Figure 2. DCs conserve T-cell stimulation efficacy when exposed to cholesterol in vitro. René R. S. Packard et al. Circ Res. 2008; 103: 965 -973 Copyright © American Heart Association, Inc. All rights reserved.
Figure 3. Systemic inflammatory response and DC lipid accumulation in vivo during hypercholesterolemia. René R. S. Packard et al. Circ Res. 2008; 103: 965 -973 Copyright © American Heart Association, Inc. All rights reserved.
Figure 4. DCs maintain T-cell priming potency in vivo during hypercholesterolemia. René R. S. Packard et al. Circ Res. 2008; 103: 965 -973 Copyright © American Heart Association, Inc. All rights reserved.
Figure 5. DCs conserve complete antigen-processing and T-cell priming capabilities within lymph nodes under in vivo hypercholesterolemia. René R. S. Packard et al. Circ Res. 2008; 103: 965 -973 Copyright © American Heart Association, Inc. All rights reserved.
Figure 6. Preserved DC machinery involved in antigen processing and T-cell stimulation during hypercholesterolemia in vivo. René R. S. Packard et al. Circ Res. 2008; 103: 965 -973 Copyright © American Heart Association, Inc. All rights reserved.
Figure 7. Preserved CD 4+ T-cell priming in vivo under dyslipidemic conditions. René R. S. Packard et al. Circ Res. 2008; 103: 965 -973 Copyright © American Heart Association, Inc. All rights reserved.